Malaria ist eine lebensbedrohlich Parasitenerkrankung, die durch Plasmodien hervorgerufen und durch die Anopheles Mücke übertragen wird. Die Ausprägung der Erkrankung wird durch verschiedene genetische Faktoren beeinflusst.
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Louicharoen C et al. (2009) Positively selected G6PD-Mahidol mutation reduces Plasmodium vivax density in Southeast Asians. |
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Timmann C et al. (2012) Genome-wide association study indicates two novel resistance loci for severe malaria. |
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Cserti-Gazdewich CM et al. (2012) Cytoadherence in paediatric malaria: ABO blood group, CD36, and ICAM1 expression and severe Plasmodium falciparum infection. |
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Turner L et al. (2013) Severe malaria is associated with parasite binding to endothelial protein C receptor. |
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Cockburn IA et al. (2004) A human complement receptor 1 polymorphism that reduces Plasmodium falciparum rosetting confers protection against severe malaria. |
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37. |
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Knight JC et al. (1999) A polymorphism that affects OCT-1 binding to the TNF promoter region is associated with severe malaria. |
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Khor CC et al. (2007) A Mal functional variant is associated with protection against invasive pneumococcal disease, bacteremia, malaria and tuberculosis. |
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Hill AV et al. (1992) Molecular analysis of the association of HLA-B53 and resistance to severe malaria. |
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Sjöberg K et al. (1992) Genetic regulation of human anti-malarial antibodies in twins. |
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Hill AV et al. (1991) Common west African HLA antigens are associated with protection from severe malaria. |
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49. |
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Kun JF et al. (1998) Polymorphism in promoter region of inducible nitric oxide synthase gene and protection against malaria. |
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Allen SJ et al. (1997) alpha+-Thalassemia protects children against disease caused by other infections as well as malaria. |
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Fernandez-Reyes D et al. (1997) A high frequency African coding polymorphism in the N-terminal domain of ICAM-1 predisposing to cerebral malaria in Kenya. |
55. |
Williams TN et al. (1996) High incidence of malaria in alpha-thalassaemic children. |
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Serjeantson SW et al. (1994) A 3.5 kb deletion in the glycophorin C gene accounts for the Gerbich-negative blood group in Melanesians. |
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Ruwende C et al. (1995) Natural selection of hemi- and heterozygotes for G6PD deficiency in Africa by resistance to severe malaria. |
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Pasvol G et al. (1982) The interaction of malaria parasites with red blood cells. |
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Pasvol G et al. (1982) Erythrocytes deficiency in glycophorin resist invasion by the malarial parasite Plasmodium falciparum. |
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Kidson C et al. (1981) Ovalocytic erythrocytes from Melanesians are resistant to invasion by malaria parasites in culture. |
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Hadley T et al. (1983) Resistance of Melanesian elliptocytes (ovalocytes) to invasion by Plasmodium knowlesi and Plasmodium falciparum malaria parasites in vitro. |
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None (1988) Elliptocytosis, malaria, and fertility in Malaysia. |
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Orphanet article Orphanet ID 673 |
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OMIM.ORG article Omim 611162 |
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Wikipedia Artikel Wikipedia DE (Malaria) |