| Klinisch | Untersuchungsmethoden | Familienuntersuchung |
| Bearbeitungszeit | 5 Tage | |
| Probentyp | genomische DNS |
| Klinisch | Untersuchungsmethoden | Hochdurchsatz-Sequenzierung |
| Bearbeitungszeit | 25 Tage | |
| Probentyp | genomische DNS |
| Klinisch | Untersuchungsmethoden | Direkte Sequenzierung der proteinkodierenden Bereiche eines Gens |
| Bearbeitungszeit | 25 Tage | |
| Probentyp | genomische DNS |
| Klinisch | Untersuchungsmethoden | Multiplex ligationsabhängige Amplifikation |
| Bearbeitungszeit | 25 Tage | |
| Probentyp | genomische DNS |
| 1. |
Board PG et al. (1989) Isolation of a cDNA clone and localization of the human glutathione S-transferase 3 genes to chromosome bands 11q13 and 12q13-14.
|
| 2. |
Menegon A et al. (1998) Parkinson's disease, pesticides, and glutathione transferase polymorphisms.
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| 3. |
None (1998) Parkinson's disease: nature meets nurture.
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| 5. |
Board P et al. (1993) Evidence against a relationship between fatty acid ethyl ester synthase and the Pi class glutathione S-transferase in humans.
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| 6. |
Smith CM et al. (1995) Genetic and radiation-reduced somatic cell hybrid sublocalization of the human GSTP1 gene.
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| 7. |
None (1981) Biochemical genetics of glutathione-S-transferase in man.
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| 8. |
Silberstein DL et al. (1982) Gene for glutathione S-transferase-1 (GST1) is on human chromosome 11.
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| 9. |
Laisney V et al. (1983) [Localization of the LDHA-GST3-ESA4 synthetic group on human chromosome 11. Analyses of the classic man-rodent hybrids and of a new type (not adhering to the wall)].
|
| 10. |
Suzuki T et al. (1984) Glutathione-S-transferase gene mapped to chromosome 11 is GST3 not GST1.
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| 11. |
Laisney V et al. (1984) Human genes for glutathione S-transferases.
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| 12. |
Kano T et al. (1987) Structure and expression of a human class pi glutathione S-transferase messenger RNA.
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| 13. |
Beutler E et al. (1988) Erythrocyte glutathione S-transferase deficiency and hemolytic anemia.
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| 14. |
Moscow JA et al. (1988) Isolation of the human anionic glutathione S-transferase cDNA and the relation of its gene expression to estrogen-receptor content in primary breast cancer.
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| 15. |
Islam MQ et al. (1989) Chromosomal localization of human glutathione transferase genes of classes alpha, mu and pi.
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| 16. |
Zusterzeel PL et al. (1999) Glutathione S-transferase isoenzymes in decidua and placenta of preeclamptic pregnancies.
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| 17. |
Kingsley DM et al. (1989) A molecular genetic linkage map of mouse chromosome 9 with regional localizations for the Gsta, T3g, Ets-1 and Ldlr loci.
|
| 18. |
Morrow CS et al. (1989) Structure of the human genomic glutathione S-transferase-pi gene.
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| 19. |
Moscow JA et al. (1989) Expression of anionic glutathione-S-transferase and P-glycoprotein genes in human tissues and tumors.
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| 20. |
Konohana A et al. (1990) Placental glutathione-S-transferase-pi mRNA is abundantly expressed in human skin.
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| 21. |
Bora PS et al. (1991) Molecular cloning, sequencing, and expression of human myocardial fatty acid ethyl ester synthase-III cDNA.
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| 22. |
Wilk JB et al. (2006) Herbicide exposure modifies GSTP1 haplotype association to Parkinson onset age: the GenePD Study.
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| 23. |
Seldin MF et al. (1991) A proximal mouse chromosome 9 linkage map that further defines linkage groups homologous with segments of human chromosomes 11, 15, and 19.
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| 24. |
Gilliland FD et al. (2004) Effect of glutathione-S-transferase M1 and P1 genotypes on xenobiotic enhancement of allergic responses: randomised, placebo-controlled crossover study.
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| 25. |
Board PG et al. (1992) The human Pi class glutathione transferase sequence at 12q13-q14 is a reverse-transcribed pseudogene.
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| 26. |
Rochelle JM et al. (1992) A linkage map of mouse chromosome 19: definition of comparative mapping relationships with human chromosomes 10 and 11 including the MEN1 locus.
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| 27. |
Zusterzeel PL et al. (2002) Paternal contribution to the risk for pre-eclampsia.
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| 28. |
Allan JM et al. (2001) Polymorphism in glutathione S-transferase P1 is associated with susceptibility to chemotherapy-induced leukemia.
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| 29. |
Henderson CJ et al. (2000) Increased resistance to acetaminophen hepatotoxicity in mice lacking glutathione S-transferase Pi.
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