| Klinisch | Untersuchungsmethoden | Familienuntersuchung |
| Bearbeitungszeit | 5 Tage | |
| Probentyp | genomische DNS |
| Klinisch | Untersuchungsmethoden | Hochdurchsatz-Sequenzierung |
| Bearbeitungszeit | 25 Tage | |
| Probentyp | genomische DNS |
| Klinisch | Untersuchungsmethoden | Direkte Sequenzierung der proteinkodierenden Bereiche eines Gens |
| Bearbeitungszeit | 25 Tage | |
| Probentyp | genomische DNS |
| Klinisch | Untersuchungsmethoden | Multiplex ligationsabhängige Amplifikation |
| Bearbeitungszeit | 25 Tage | |
| Probentyp | genomische DNS |
| 1. |
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| 4. |
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| 5. |
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| 8. |
None (1985) The isoenzymes of glutathione transferase.
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| 9. |
Strange RC et al. (1985) The human glutathione S-transferases: developmental aspects of the GST1, GST2, and GST3 loci.
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| 10. |
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| 11. |
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| 12. |
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| 13. |
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| 14. |
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| 15. |
None (1996) Chemoprevention of lung cancer by isothiocyanates.
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| 16. |
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| 17. |
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| 18. |
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| 19. |
Zhong S et al. (1992) Chromosomal assignment and linkage analysis of the human glutathione S-transferase mu gene (GSTM1) using intron specific polymerase chain reaction.
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| 20. |
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| 21. |
Islam MQ et al. (1989) Chromosomal localization of human glutathione transferase genes of classes alpha, mu and pi.
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| 22. |
None (1981) Biochemical genetics of glutathione-S-transferase in man.
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| 23. |
Cotton SC et al. (2000) Glutathione S-transferase polymorphisms and colorectal cancer: a HuGE review.
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| 24. |
Godschalk RW et al. (2001) Modulation of DNA and protein adducts in smokers by genetic polymorphisms in GSTM1,GSTT1, NAT1 and NAT2.
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| 25. |
Lee KA et al. (2001) Increased frequencies of glutathione S-transferase (GSTM1 and GSTT1) gene deletions in Korean patients with acquired aplastic anemia.
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| 26. |
Carless MA et al. (2002) The GSTM1 null genotype confers an increased risk for solar keratosis development in an Australian Caucasian population.
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| 27. |
Verlaan M et al. (2003) Glutathione S-transferase Mu null genotype affords protection against alcohol induced chronic pancreatitis.
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| 28. |
Lohmueller KE et al. (2003) Meta-analysis of genetic association studies supports a contribution of common variants to susceptibility to common disease.
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| 29. |
Mannervik B et al. (1992) Nomenclature for human glutathione transferases.
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| 30. |
French D et al. (2005) Global gene expression as a function of germline genetic variation.
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| 31. |
Brennan P et al. () Effect of cruciferous vegetables on lung cancer in patients stratified by genetic status: a mendelian randomisation approach.
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| 32. |
van Poppel G et al. (1992) Increased cytogenetic damage in smokers deficient in glutathione S-transferase isozyme mu.
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| 33. |
Huang RS et al. (2009) Population-specific GSTM1 copy number variation.
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| 34. |
DeJong JL et al. (1991) The human Hb (mu) class glutathione S-transferases are encoded by a dispersed gene family.
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| 35. |
Strange RC et al. (1991) The human glutathione S-transferases: a case-control study of the incidence of the GST1 0 phenotype in patients with adenocarcinoma.
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