Clinical feature: 

Definition: Glomerulonephritis as an immunological disease often has a genetic background that can be determined.

Literature: 

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Berger J et al. (1969) IgA glomerular deposits in renal disease.
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Ault BH et al. (1997) Human factor H deficiency. Mutations in framework cysteine residues and block in H protein secretion and intracellular catabolism.
Brai M et al. (1988) Combined homozygous factor H and heterozygous C2 deficiency in an Italian family.
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Fijen CA et al. (1996) Heterozygous and homozygous factor H deficiency states in a Dutch family.
Hegasy GA et al. (2002) The molecular basis for hereditary porcine membranoproliferative glomerulonephritis type II: point mutations in the factor H coding sequence block protein secretion.
Høgåsen K et al. (1995) Hereditary porcine membranoproliferative glomerulonephritis type II is caused by factor H deficiency.
Levy M et al. (1986) H deficiency in two brothers with atypical dense intramembranous deposit disease.
Licht C et al. (2006) Deletion of Lys224 in regulatory domain 4 of Factor H reveals a novel pathomechanism for dense deposit disease (MPGN II).
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McRae JL et al. (2002) Location and structure of the human FHR-5 gene.